GTD2B Rabbit Polyclonal Antibody, 50ul Cell Separation and Collection with deletion of the Golgi

with deletion of the Golgi and endoplasmic reticulum signals resulting in abnormal subcellular localization

TLRs are highly conserved from Drosophila to humans and share structural and functional similarities

setting in motion a cascade of downstream signals

Mutation in this gene results in deletion of the DAG binding-like motif and causes a recessive ataxia

The encoded protein also enhances Akt signaling by mediating phosphoinositide 3-kinase (PI3K)-dependent activation of Akt by growth factor receptor tyrosine kinases and G protein-coupled receptors

GTD2B Rabbit Polyclonal Antibody, 50ul Cell Separation and Collection with deletion of the GolgiThis gene encodes a glycosylated phosphoprotein with a leucine zipper motif two helix loop helix motifs (I repeats) that are similar to domains found in the TFII I family of transcription factors one CHARLIE8 transposable element like sequence and a BED zinc finger. This gene lies within a region that is deleted in Williams Beuren syndrome. Alternatively spliced variants which encode different protein isoforms have been described; however not all